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The mission of the als association alsa ; is to discover the cause and cure for amyotrophic lateral sclerosis lou gehrig's disease ; through dedicated research and provide patient services, education for health care professionals and the general public, and advocacy for als research and health-related issues. Numerous therapeutic options are available to treat acute symptoms, induce remission, and establish a maintenance regimen, for example, sinus infection cipro.
McCleskey 30 ; have suggested that there are at least two high affinity calcium binding sites on the channel and that calcium binding to one of these sites on the extracellular side of the channel ; provides electrostatic repulsion of calcium bound to another calcium binding site presumably deeper within the channel ; to facilitate ion permeation through a multi-ion single file pore. Kostyuk et al. 31 ; have postulated high affinity calcium-magnesium p&. 6.6, PKM~ 4.2 ; binding sites on the extracellular side of the calcium channel which controls the structure and ion selectivity of the channel. It is possible that these extracellular high affinity calcium binding sites which regulate DHP binding are the same calcium binding sites postulated by Kostyuk et al. 31 ; to control the ion selectivity of the calcium channel or by Hess and Tsien 29 ; to facilitate electrostatic repulsion of calcium through a multi-ion single file pore. Consistent with this, DHP binding to skeletal muscle channels occurs with high affinity pI& 6.5 ; over a broad range of calcium 3 log units ; and with negative cooperativity, indicating that several classes of high affinity calcium sites may be present on the calcium channels. Magnesium also increases DHP binding to partially purified calcium channels, although it is approximately 500-fold less effective than calcium PK~.~ 3.9 ; . Further, in both heart and skeletal muscle, saturating calcium is capable of supporting lo-20% more PN200-110 binding than saturating magnesium Figs. 2 and 3 ; . This suggests that some component of DHP binding may be calcium-specific. Our studies indicate that similar high affinity calcium binding sites exist on skeletal muscle and cardiac muscle calcium channels and that these sites allosterically regulate DHP binding. It is likely that calcium or higher concentrations of magnesium ; result in the conversion from a low affinity DHP binding state that might not be detected under the conditions of the binding assay ; to a high affinity DHP binding state. Similar conclusions have been drawn from DHP binding to brain, heart, and smooth muscle calcium channels 15-17 ; . Since calcium binding enhances dihydropyridine binding in all of these tissues, then by reciprocity, DHP binding should enhance calcium binding. If this is the case, then dihydropyridines could perhaps block calcium channels by increasing calcium or magnesium ; affinity and slowing calcium flux though the ion pore as we 32 ; and Glossmann et al. 17 ; have previously suggested. If DHP increases the calcium affinity of the calcium channel it is possible that this could result in calcium binding so tightly that it could not pass through a multi-ion single file ion pore.

Pharmaceutical compositions containing them, a process for preparing the pharmaceutical compositions, and their use in therapy. In formula I ; D represents CH2 or CH2CH2, E represents C O ; NH NHC O ; and R3 represents a group of formula I, for instance, cipro and flagyl. FIGURE 5 Reciprocal of the K + influx dependence on the extracellular H + concentration Dixon plot ; . Fluxes were measured in solutions containing choline-Cl and sucrose choline-Cl: sucrose, in mM ; : 145: 0 0 50: 164 O 20: 215.5 O 0: 250 U ; . K influx was measured in the presence of inhibitors 0.1 mM ouabain, bumetanide, EGTA ; . Flux solutions contained 7.5 mM KCI, 10 mM glucose, 10 mM MES, and 10 mM TRIS. pH was adjusted to 7.2, 7.5, 8.0, or 9.0 with choline-OH. Results represent measurements from at least three individual experiments three different blood donors, triplicate samples.

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These drugs are typically prescribed with either continuous daily ; dosing throughout the month or an intermittent dosing regimen. 2. Daily users of alcohol. Daily ingestion of alcohol may be associated with a higher incidence of isoniazid hepatitis. 3. Patients with current chronic liver disease or severe renal dysfunction. Pyrazinamide. Pyrazinamide inhibits renal excretion of urates, frequently resulting in hyperuricemia which is usually asymptomatic. If hyperuricemia is accompanied by acute gouty arthritis, RIFATER, because it contains pyrazinamide, should be discontinued. Information for Patients Food Interactions: Because isoniazid has some monoamine oxidase inhibiting activity, an interaction with tyramine-containing foods cheese, red wine ; may occur. Diamine oxidase may also be inhibited, causing exaggerated response eg, headache, sweating, palpitations, flushing, hypotension ; to foods containing histamine eg, skipjack, tuna, other tropical fish ; . Tyramine- and histamine-containing foods should be avoided in patients receiving RIFATER. RIFATER, because it contains rifampin, may produce a reddish coloration of the urine, sweat, sputum, and tears, and the patient should be forewarned of this. Soft contact lenses may be permanently stained. Patients should be instructed to take RIFATER either 1 hour before or 2 hours after a meal. Patients should be instructed to notify their physicians promptly if they experience any of the following: fever, loss of appetite, malaise, nausea and vomiting, darkened urine, yellowish discoloration of the skin and eyes, pain or swelling of the joints. Compliance with the full course of therapy must be emphasized, and the importance of not missing any doses must be stressed. Laboratory Tests A complete blood count CBC ; , liver function tests, and blood uric acid determinations should be obtained prior to instituting therapy and periodically throughout the course of therapy. Because of a possible transient rise in transaminase and bilirubin values, blood for baseline clinical chemistries should be obtained before RIFATER dosing. Drug Interactions Rifampin. Enzyme Induction: Rifampin is known to induce certain cytochrome P-450 enzymes. Coadministration of RIFATER, because it contains rifampin, with drugs that undergo biotransformation through these metabolic pathways may accelerate elimination. To maintain optimum therapeutic blood levels, dosages of drugs metabolized by these enzymes may require adjustment when starting or stopping concomitantly administered rifampin. Rifampin has been reported to accelerate the metabolism of the following drugs: anticonvulsants eg, phenytoin ; , antiarrhythmics eg, disopyramide, mexiletine, quinidine, tocainide ; , anticoagulants, antifungals eg, fluconazole, itraconazole, ketoconazole ; , barbiturates, beta-blockers, calcium channel blockers eg, diltiazem, nifedipine, verapamil ; , chloramphenicol, ciprofloxacin, corticosteroids, cyclosporine, cardiac glycoside preparations, clofibrate, oral contraceptives, dapsone, diazepam, haloperidol, oral hypoglycemic agents sulfonylureas ; , methadone, narcotic analgesics, nortriptyline, progestins, and theophylline. It may be necessary to adjust dosages of these drugs if they are given concurrently with RIFATER since it contains rifampin. Rifampin has been observed to increase the requirements for anticoagulant drugs of the coumarin type. In patients receiving anticoagulants and RIFATER concurrently, it is recommended that the prothrombin time be performed daily or as frequently as necessary to establish and maintain the required dose of anticoagulant and climara.

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The first molecular analysis of the ligand binding-negative phenotype was reported by Ritchie et al. 147 ; . These investigators used PCR to amplify exons of the VDR gene in three related HVDRR patients from families F18, F34, and F36 16, 17, 25, ; . A single nucleotide base change was identified in the codon for tyrosine TAC ; at amino acid 295, which changed the sequence to an ochre termination codon TAA ; 147 ; . The same mutation was found in each patient. The Tyr295 stop mutation truncates 132 aa of the carboxy terminus of the VDR, which results in the deletion of a major portion of the LBD, thereby creating the ligand bindingnegative phenotype. The reconstructed mutant VDR exhibited no specific [3H]1, 25- OH ; 2D3 binding and failed to activate gene transcription. The Tyr295 stop mutation was the first mutation identified in the VDR LBD. The location of this mutation and other LBD mutations is shown in Fig. 13 and summarized in Table 4. Four additional families F35, F37, F38, and F39 ; related to the families described above make up a large kindred in which consanguineous marriages occurred. The entire kindred was fully analyzed by Malloy et al. 22 ; see Fig. 2 ; . A total of eight children from this kindred exhibited HVDRR with alopecia and the same hormone binding-negative HVDRR phenotype. All of the affected children were homozygous for the ochre stop mutation at Tyr295, and their parents were heterozygous as determined by analysis of a RsaI RFLP created by the mutation 22 ; . Interestingly, the 30, 000 mol wt truncated protein that is predicted to be produced by this mutation could not be detected by Western blot analysis. In addition, Northern blot analysis of RNA obtained from skin fibroblasts or EBV-transformed lymphoblasts from the patients failed to detect any mutant VDR mRNA in all but one case F35 ; . The absence of mRNA transcripts has been reported in other genetic diseases where a premature stop mutation has been found and, in this case, likely accounts for the absence of the mutant-truncated VDR protein 255257. The health of the liver. Measuring HBV viral load, liver enzyme levels, and AFP in the blood cannot determine if and how much liver damage exists. For this, a liver biopsy is needed. Liver biopsies are only recommended for patients who have a high HBV viral load above 100, 000 ; and elevated liver enzymes. A liver biopsy is usually performed on an outpatient basis in a hospital. Sometimes, a trained healthcare provider such as a hepatologist or a gastroenterologist can perform a liver biopsy in his or her office. An ultrasound is some10 and clonazepam. Tance. For this reason we supplemented our studies by Ussing chamber experiments. This technique allows the measurement of transepithelial membrane currents Isc ; , although, due to imperfect edge sealing, the absolute magnitudes of Vte and Rte are certainly underestimates. The results obtained by both methods match closely, indicating that the Vm values observed in the impalement studies represent corresponding conductance changes. Enhanced ENaC activity in parallel with an impaired cAMP-activated Cl conductance are pathophysiological hallmarks of CF in airway function. Since mutations of CFTR are the cause for CF, enhanced Na conductance in CF airways should somehow be related to CFTR and its mutations as they occur in CF. Several recent reports focus on this unresolved issue and demonstrate that CFTR, when activated by cAMP, downregulates ENaC activity 3, 1113 ; . Although this reciprocal regulation of CFTR and ENaC has been demonstrated in various types of epithelial and nonepithelial cells, it has not been shown for respiratory epithelial cells, where it probably plays an important role in the regulation of ion transport and thus may have a major impact on the pathophysiology of CF. These data indicate that stimulation of the airways by secretagogues leads to activation of CFTR and an increase of apical Cl conductance. This is probably of specific importance for the serous type of submucosal gland cells, as expression of CFTR is predominant in these cells 17, 18 ; . These cells are probably the target of stimulation and secrete electrolytes. In contrast, surface epithelial cells expressing ENaC and very limited amounts of CFTR are thought to be responsible for absorption of electrolytes 19 ; . Given the fact that both ENaC and CFTR are coexpressed in the same cell, Na conductance will be turned off by the activation of CFTR and the cross-talk between CFTR and ENaC, with the consequence of a decrease of electrolyte absorption or maybe even a switching to NaCl secretion. Taken together, inhibition of ENaC by CFTR in the airway epithelium may reflect an important mechanism by.

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Ms Cynthia Plaaitjies comes to the pharmacy w ith the follow ing prescription Theophen Elixir 15ml 3 times a day Amoxycillin 500mg 3 times a day Flagyl 400mg 12 hourly for 7 days Ciprobay 500mg stat Doxycycline 100mg 12 hourly for 7 days Dolorol 2 prn Age - 23 Diagnosis - STI Influenza Would you be happy to dispense this prescription? Give reasons for your answer. 4 and clonidine.

The level of the medication in the blood should be monitored carefully and adjusted so that the least amount possible is used to be effective. Docusate Sod Cap 100mg Dioctyl Cap 100mg Fletchers' Enemette Microenema 5ml Norgalax Micro-Enem 120mg 10g Tube Docusol Adult Soln 50mg 5ml S F Co-Danthrusate Cap 50mg 60mg Co-Danthrusate Susp 50mg 60mg 5ml S F Glycerol Suppos Infant's 1g ; Glycerol Suppos Child 2g ; Glycerol Suppos Adult's 4g ; Senna Tab 7.5mg Senna Gran Standardised 15mg 5ml Senna Oral Soln 7.5mg 5ml Ispaghula Senna Fruit Gran 54.2% 12.4% Senokot Gran Senokot Syr 7.5mg 5ml Manevac Gran Manevac Sach 4g Sod Picosulf Elix 5mg 5ml S F Ciprofibrate Tab 100mg Modalim Tab 100mg Acipimox Cap 250mg Atorvastatin Tab 10mg Atorvastatin Tab 20mg Atorvastatin Tab 40mg Atorvastatin Tab 80mg Lipitor Tab 10mg Lipitor Tab 20mg Lipitor Tab 40mg Lipitor Tab 80mg Cerivastatin Tab 100mcg Cerivastatin Tab 200mcg Lipobay Tab 100mcg Bezafibrate Tab 200mg Bezafibrate Tab 400mg M R Bezalip Tab 200mg and combivent.

Where sjt Ik ; sjt Ij t ; , Ik ; - sjt I p , Ik ; Intuitively, sjt Ik ; is the change in the probability j of choosing j when a physician switches his her information set for drug j from I p to conditional on him her knowing Ik Ik t ; Equation 17 ; shows that the marginal k return of detailing depends on sjt Ik t and sjt I p ; , which are weighted by Mkt and 1 - Mkt , k respectively. This weighted average is further adjusted by Mjt Djt . It is worth noting that Sjt Djt increases decreases ; with Mkt if sjt Ik t - sjt I p is positive negative ; . k Consider a situation where a new drug enters a market with a matured incumbent in the sense that the representative opinion leader has learnt the true quality of the incumbent, i.e., Ik t ; Ik . Conditional on M , equations 16 ; and 17 ; imply that the entrant's marginal return of detailing will increase with its market share. Moreover, the detailing elasticity of demand in our model could increase or decrease over time partly depending on how I t ; evolves. In particular, even after the uncertainty about the drug quality is completely resolved, detailing still affects demand as long as I 0, and its effect depends on I t ; , and Mjt-1 i.e., GI ; . On the contrary, previous models of learning and detailing advertising, which follow jt-1 the framework of Erdem and Keane 1996 ; , imply that the detailing advertising elasticity of demand diminishes over time as uncertainty about product quality is slowly resolved. This 15, for example, cipro treatment. La direttiva 89 552 CEE del Consiglio relativa al coordinamento di determinate disposizioni legislative, regolamentari e amministrative degli Stati membri concernenti l'esercizio delle attivit televisive "Servizi di media audiovisivi senza frontiere" ; COM 2007 ; 0170 Decisione: trasmettere all'on. Hieronymi e ai relatori ombra. 1.11 Comunicazione della Commissione al Parlamento europeo, al Consiglio, al Comitato economico e sociale e al Comitato delle regioni: La regolamentazione europee e i mercati europei delle comunicazioni elettroniche 2006 dodicesima relazione ; COM 2007 ; 0155 Merito: ITRE Decisione: nessun parere. 1.12 Comunicazione della Commissione al Parlamento europeo, al Consiglio, al Comitato economico e sociale e al Comitato delle regioni: i2010 Relazione annuale sulla societ dell'informazione 2007 COM 2007 ; 0146 Decisione: trasmettere all'on. Decamps per informazione. 1.13 Comunicazione della Commissione al Consiglio, al Parlamento europeo, al Comitato economico e sociale europeo e al Comitato delle regioni - Migliorare il trasferimento delle conoscenze tra gli organismi di ricerca e le imprese nell'insieme dell'Europa: per un'innovazione aperta Attuare l'agenda di Lisbona COM 2007 ; 0182 Decisione: PSE. 1.14 Libro verde. Accesso del pubblico ai documenti detenuti dalle istituzioni della Comunit europea Esame della situazione COM 2007 ; 0185 Decisione: pendente. 1.15 Seguito della risoluzione del Parlamento europeo del 13 dicembre 2006 sul programma legislativo e di lavoro della Commissione per il 2007 SP 2007 ; 498 3 ; Decisione: da discutere nel corso delle prossime riunioni con i commissari Figel', Reding e Wallstrm. 1.16 Osservazioni concernenti il sistema educativo: le universit a Cipro Decisione: da discutere nel corso della prossima riunione con il commissario Figel'. 2. Nomina di esperti per la capitale europea della cultura 2011 Decisione: presentare alla commissione la proposta del presidente per approvazione. 3. Forum della giovent dell'Unione europea Decisione: i coordinatori sono informati in merito alla lettera del vicepresidente VidalQuadras. 4. Progetto "La Fenice Libert e liberazioni" Decisione: i gruppi dovranno verificare se i membri sono disponibili a partecipare il 25 maggio 2007 venerd della settimana di tornata ; . 5. "Premio europeo del libro and coumadin. Note: Quinolones are not approved by the Food and Drug Administration FDA ; for the treatment of children because of the potential for retarded skeletal growth. However, some pediatricians prescribe pyrazinamide and ciprofloxacin or ofloxacin in TST-positive children who are close contacts of an MDR-TB patient see Option II above ; . The use of quinolones in children is being re-reviewed by the FDA and may be approved in the future.

D42 - Treatment access and availability CDD1105 - PLWHA involvement in health care provision - The Thai experience A. Kumpipak Medcines sans Frontieres, Nursing, Bangkok, Thailand Issues: Comprehensive Continum Care CCC ; Centres -Thai PLWHA involvement in the public health system Description: TNP + has been instrumental in advocating for PLWHA inclusion and involvement in health care provision. TNP + along with its partners, MSF- Belgium and AIDS ACCESS Foundation decided to develop the role of PLWHA as partners in the provision of comprehensive and continuous care by allowing PLWHA to provide treatment support. This has included psychological support, counseling, home visits and adherence to treatment support. They now work together in numerous hospitals Thailand wide and together, with a multi disciplinary team comprising doctors, nurses, pharmacists and laboratory technicians form the CCC Centres which provide ARV. TNP + provides training and support for the PLWHA who work in these centres in order for them to carry out their duties professionally and competently. Incorporating PLWHAs role into the public health system has ensured sustainability and has been instrumental in building PLWHAs confidence and pride, enabled them to take an active role in their own health and gained them greater respect and admiration from the staff working in the public health system. It has also helped prepare the public health system for the care and treatment of PLWHA and ARV provision. The last few years has seen Thailands ARV program grow enormously from 1500 PLWHA receiving treatment through drug trials in 2002 to present day where more than 80, 000 PLWHA receive ARV through the public health system. Lessons learned: PLWHA need to lobby their respective governments to provide valuable and meaningful roles for PLWHA within the public health system. Recommendations: Every country can learn from Thailand of the integral and vital role that PLWHA can perform in health care provision. We hope and recommend that our audiences learn more about the Thai CCC model and consider replicating it for use in theirown countries and cozaar.

B. Seol: Comparative in vitro activities of enrofloxacin, ciprofloxacin and marbofloxacin against Staphylococcus intermedius isolated from dogs.
Eriksson et al. Ciprofloxacin-treated human lymphocytes induce gene programs and cyclobenzaprine. Ciprofloxacin 500mg oral twice daily Ciprofloxacin 500mg oral twice daily children: 15mg kg oral twice daily ; or Doxycycline 100mg twice daily 2.2mg kg oral twice daily. The foremost objective in veterinary medicine is patient welfare. Ideally, sound veterinary medicine is effective, safe, proven and holistic in that it considers all aspects of the animal patient in the context of its environment. Diagnosis should be based on sound, accepted principles of veterinary medicine. Proven treatment methods should be discussed with the owner or authorized agent when presenting the treatment options available. Informed consent should be obtained prior to initiating any treatment, including CAVM. Clients usually choose a medical course of action on the advice of their veterinarian. Recommendations for effective and safe care should be based on available scientific knowledge and the medical judgment of the veterinarian and depakote and cipro, for example, cipro side effects. Bactroban xanax withdrawal bactroban buy bactroban bactroban xanax withdrawal bactroban buy bactroban cns adderall concerta provigil ritalin strattera antidepressants amitriptyline celexa effexor xr elavil lexapro lithium paxil prozac remeron wellbutrin zoloft antibiotics medications amoxicillin augmentin bactrim biaxin cephalexin cipro doxycycline erythromycin keflex levaquin penicillin zithromax antiviral acyclovir amantadine tamiflu valtrex nerve pills alprazolam ativan buspar clonazepam diazepam klonopin lorazepam oxazepam rivotril valium xanax arthritis medications bextra lodine voltaren asthma treatment foradil birth control medications alesse mircette ortho evra ortho tricyclen ortho tricyclen lo plan b triphasil yasmin blood pressure medication aceon atenolol norvasc cancer medications femara cholesterol treatment crestor lipitor vytorin zocor diabetic avandamet insulin metformin stomach aciphex bentyl detrol la prevacid prilosec protonix ranitidine hcl hair losstreatment propecia blood thinners coumadin plavix eerectile dysfunction medications cialis levitra viagra migraines headache treatment butalbital esgic plus fioricet imitrex imitrex oral muscle pain carisoprodol flexeril skelaxin soma zanaflex pain medication codeine darvocet hydrocodone lorcet lortab norco oxycodone percocet tramadol ultram vicodin vicoprofen zydone schizophrenia meds abilify zyprexa seizures medication neurontin topamax sexual health medications acyclovir aldara condylox famvir valtrex skin care treatment accutane aphthasol atarax lamisil metronidazole nizoral protopic renova retin-a sumycin tretinoin sleeping pills ambien rozerem sonata quit smoking zyban thyroid hormonal treatment levothyroxine synthroid appetite suppressants adipex bontril didrex diethylpropion ionamin meridia phendimetrazine phentermine tenuate xenical a mupirocin topical ; mupirocin myoo-peer-oh-sin ; is used to treat bacterial infections. Figure 2. Comparison of fibrinolytic activity of smokers and nonsmokers. The dilute blood clot lysis time is plotted as a reciprocal function 106 lysis time in minutes ; 2 ; . Euglobulin activity is plotted as the average diameter of lysed zones mm ; in fibrin plates after 17 hours incubation at 37 C. The means are indicated by horizontal bars and the standard deviations by dotted lines and detrol.
Table selected baseline and demographic characteristics n 32.
XML Namespaces XML Namespaces are used to support the association of specific XML markup to a particular vocabulary and schema. Namespaces allow the mixture of XML markup from different vocabularies. In fact, using SOAP and XML Schemas require the use of namespaces and at least two vocabularies. An XML Namespace is a unique identifier specified using a URI uniform resource identifier ; . Thus, a namespace identifier looks like a URL or a web page address. However, an XML Namespace identifier is not a web page address and there is no expectation of finding any resource at the URI that is used. The combination of the unique domain name and the URI path establish the uniqueness that is required of namespace identifiers. The use of XML Namespaces as shown later in this document are required. Failure to use the proper namespace will result in an XML validation failure and an error response. More information on XML Namespaces can be found in the XML references already cited. The drug resistant pattern of isolated organisms is shown in Table 6. It shows that Staph aureus was sensitive to ciprofloxacin, erythromycin, cotrimoxazole and cloxacillin 100% of the isolates in treatment Group I. Only one isolate was sensitive to ampicillin. However in the same treatment group, Pseudomonas species was most of the time resistant to the common oral antibiotics. The safety and efficacy of ciprofloxacin extended-release tablets in treating infections other than urinary tract infections has not been demonstrated. This whole approach to prevention involves treating risk as a disease, which means a lot more people will be getting a lot more drugs, and we don't know the long-term results of this and claritin.
Levofloxacin has been studied in aureus pyelonephritis in mice, and shown to be more active than ciprofloxacin in vitro figure ; 4.
00093086301 00093086401 00093718101 CIPROFLOXACN TAB 250MG CIPROFLOXACN TAB 500MG OFLOXACIN TAB 300MG OFLOXACIN TAB 400MG CIPROFLOXACN TAB 250MG CIPROFLOXACN TAB 500MG CIPROFLOXACN TAB 750MG CIPROFLOXACN TAB 250MG CIPROFLOXACN TAB 500MG CIPROFLOXACN TAB 500MG CIPROFLOXACN TAB 750MG CIPROFLOXACN SUS 5% CIPROFLOXACN SUS 10% CIPROFLOXACN TAB 250MG CIPROFLOXACN TAB 500MG CIPROFLOXACN TAB 750MG CIPROFLOXACN TAB 250MG CIPROFLOXACN TAB 500MG CIPROFLOXACN TAB 750MG CIPROFLOXACN TAB 250MG CIPROFLOXACN TAB 500MG CIPROFLOXACN TAB 500MG CIPROFLOXACN TAB 750MG OFLOXACIN TAB 300MG OFLOXACIN TAB 400MG CIPROFLOXACN TAB 500MG CIPROFLOXACN TAB 250MG 0 0 0 570 1, 549 0 0 ##TEXT##.00 ##TEXT##.00 ##TEXT##.00 ##TEXT##.00 ##TEXT##.00 ##TEXT##.00 ##TEXT##.00 ##TEXT##.00 ##TEXT##.00 ##TEXT##.00 ##TEXT##.00 6.64 , 415.84 ##TEXT##.00 ##TEXT##.00 ##TEXT##.00 , 320.93 2, 141.69 , 421.35 ##TEXT##.00 ##TEXT##.00 ##TEXT##.00 ##TEXT##.00 ##TEXT##.00 9.19 ##TEXT##.00 ##TEXT##.00 0.00% 0.00% 0.00% 0.00% 0.00% 0.00% 0.00% 0.00% 0.00% 0.00% 0.00% 0.03% 0.12% 0.00% 0.00% 0.00% 6.64% 18.05% 0.56% 0.00% 0.00% 0.00% 0.00% 0.00% 0.08% 0.00% 0.00.

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